Thursday, November 23, 2017
Wednesday, November 22, 2017
How efforts to reverse climate change could Destroy the planet in catastrophic backfire, scientists warn
Human efforts to reverse climate change could DESTROY the planet in catastrophic backfire, scientists warn
Tuesday, November 21, 2017 by: Isabelle Z.
Tags: backfire, climate change, geoengineering, Global Cooling, global warming, sulfur dioxide, volcanic eruptions, weird science
Tags: backfire, climate change, geoengineering, Global Cooling, global warming, sulfur dioxide, volcanic eruptions, weird science
(Natural News) Reversing climate change seems to be the celebrity cause du jour, and there are many people who feel that significant action is needed to help counteract global warming. Most people want to protect the planet on which we live, but there is a lot of misinformation out there and plenty of debate on the best way to achieve this.
Some have touted geoengineering, or the purposeful manipulation of our climate to reverse its damages, as a way of dealing with climate change. However, experts have warned that some caution is needed here. Trying to manipulate the environment could backfire in spectacular fashion on humanity, destroying the very planet that supports us and that we are trying to save.
According to a new study in Nature Communications, trying to cool the planet artificially to counteract human-caused global warming is going to end badly for us.
The study was carried out by researchers from the University of Exeter, and their findings were very disturbing. For their research, they enlisted the help of advanced climate models to simulate how proposed climate interventions like intentionally injecting aerosols into our atmosphere to reduce the sun’s power to fuel destructive storms could affect us.
This might seem like a good idea on the surface. After all, volcanic eruptions do this naturally, with the particles emitted serving as a barrier to the sun’s hot rays and preventing them from reaching the planet’s surface.
This causes a short-term cooling, and some have proposed that humans attempt to mimic this in the northern hemisphere to lessen the strength of tropical storms. Emitting sulfur dioxide, just as volcanic eruptions do, has been touted as a cheap and relatively feasible way to deal with the problem. While the researchers’ climate models found that this would indeed have the potential to decrease the number of storms in the North Atlantic, it would come at a very significant cost.
Specifically, the reduction in tropical storms in this area would cause drought in some parts of Africa to skyrocket. In particular, the area between the Sudanian Savanna and the Sahara known as the Sahel would be impacted heavily. On the other hand, if aerosols were injected into the atmosphere above our planet’s southern hemisphere, drought in certain areas of Africa would actually be reduced, including the Sahel, while tropical storms in the Atlantic Ocean would rise significantly. It’s a losing proposition no matter which way you look at it.
Researchers call for regulations on geoengineering
Therefore, the researchers believe that geoengineering should be tightly controlled as a way to deal with climate change given this highly undesirable outcome. Otherwise, some countries could conceivably start carrying out geoengineering projects that would help them reach their own climate goals at the expense of other areas. In fact, it’s not a stretch to imagine that some could even do it intentionally to wreak havoc on nations they don’t agree with – or at the very least shrug off the risks because they don’t affect their country directly.
The study’s lead author, Dr. Anthony Jones, said: “Our results confirm that regional solar geoengineering is a highly risky strategy which could simultaneously benefit one region to the detriment of another.”
Moreover, the world would have to use many megatons of sulfur dioxide and pump it into the air every year to maintain the cooler temperatures needed, and it could also have unforeseen effects on water scarcity, food security and even power generation.
This really shouldn’t come as much of a surprise. Nature has shown us time and time again that humans interfere with it at their own peril. Push nature, and it will push right back and remind everyone who is really in charge.
Chuck Norris Sues for MRI Dye Injuring His Wife
- Magnetic resonance imaging (MRI) is a study allowing your physician to see detailed images of your organs and tissues using a large magnet and radio waves
- Gadolinium-based contrast agents (GBCAs) may be prescribed to enhance the image but the heavy metal gadolinium is known to deposit in your brain, bone and organs
- The FDA acknowledges the metal may be found in your tissues for years but claims there is no known effect, despite the rising number of people suffering from kidney damage, cognitive dysfunction and muscle wasting
- Strategies you may consider if an enhanced MRI is absolutely necessary include optimizing your gut and nutrition, eating fermented foods and using a sauna to aid detoxification
By Dr. Mercola
Magnetic resonance imaging (MRI) is an imaging study that allows your physician to see detailed pictures of your organs and tissues. Raymond Damadian invented the machine and performed the first total body image in 1977.1 This process has been called the one of the greatest medical breakthroughs of the 20th century.
The MRI machine uses a large magnet, radio waves and a computer to take detailed cross-sectional pictures of your internal organs and tissues.2 The scanner looks like a tube with a table that enables you to slide into the tunnel of the machine to gather data. Unlike CT scans or X-rays that use ionizing radiation known to damage DNA, the MRI uses magnetic fields.
Images from an MRI give physicians better information about abnormalities, tumors, cysts and specific organ problems with your heart, liver, uterus, kidneys and other organs. In some instances, your physician may want an enhanced MRI, one using contrast agents to improve the clarity of the images produced. In one of every three MRIs with contrast, the agent used is gadolinium.3
What Is Gadolinium?
This is a chemical contrast medium or dye that is injected to enhance the quality of the images. Gadolinium is bonded to a chelating agent that is intended to reduce the toxicity of the chemical to your body as gadolinium is a dangerous heavy metal known to cause neurological damage. There are several products that include gadolinium, called gadolinium-based contrast agents (GBCA).4
The U.S. Food and Drug Administration (FDA) released guidelines on the use of GBCAs5 as they began an investigation into the potential health effects from brain deposits of the heavy metal. The FDA acknowledged that repeated use of GBCAs may result in gadolinium deposits in your brain and other tissues but has not determined if there are any adverse health effects from these deposits.
To reduce accumulation, the FDA recommends health care professionals limit use to circumstances where additional information garnered by the use of gadolinium is absolutely necessary.6 In response, institutional review boards responsible for the safety of patients in clinical trials or research studies developed consent forms that include a list of known risks, such as kidney damage and brain accumulation, with boxed warnings not to exceed recommended dosages.7
Johns Hopkins Office of Human Subject Research acknowledges specific challenges when using enhanced MRI imaging with GBCA as "there is potential severe toxicity related to a diagnostic procedure with no direct therapeutic value."8 GBCAs are sold under several different names based on the chelating agent to which they are bound and the drug company that developed them. These include:
Physiological Issues Associated With Heavy Metal Toxicity
Heavy metals are naturally occurring elements with a high atomic weight and a density five times that of water.9 They are widely distributed throughout the environment from industrial, agricultural, medical and technical pollution. Heavy metal toxicity has documented potential for serious health consequences, including kidney, neurological, cardiovascular, skeletal and endocrine damage.10
While your body requires small amounts of some metals, such as zinc, manganese, iron and copper, high amounts of these and environmental pollutants is dangerous.11 Metals that are most commonly associated with poisoning are arsenic, lead, mercury and cadmium, which are also the heavy metals most commonly found in environmental pollution.
Symptoms of heavy metal poisoning vary based on the organ systems affected. Scientists have found that heavy metals also increase oxidative stress secondary to free radical formation.12 The health risks associated with heavy metal toxicity have proven to be a major health threat, especially those that do not have a biological role, as they often remain present in plants and animals.
Testing for heavy metal toxicity includes blood, urine and hair and nail analysis for cumulative exposure. Symptoms of accumulation of gadolinium will be related to the organ systems affected and may include nausea, vomiting, central nervous system dysfunction,13 headache and gastrointestinal disturbances.14
Chuck Norris Focuses Attention on Heavy Metal Risks After Enhanced MRI
After undergoing three MRIs with GBCAs to evaluate her rheumatoid arthritis, Gena O'Kelley, the wife of American film icon Chuck Norris,15 began experiencing severe physical symptoms that began with a burning sensation in her skin. She described it as if there was acid burning her skin, slowly covering her body.16 She reports that she visited the emergency room five or six nights in a row, while doctors ran multiple tests for ALS, MS, cancer and Parkinson's disease.
However, it was O'Kelley who made the connection between her burning skin, contracted arm and cognitive issues and the multiple MRIs she had undergone. She told Full Measure:17
"When we got to the hospital in Houston this last time, and I'm so bad and I said, listen, I am sober enough in my thinking right now, because I had such brain issues going on, I said I'm only going to be able to tell you this one time and I need you to listen to me very closely. I have been poisoned with gadolinium or by gadolinium and we don't have much time to figure out how to get this out of my body or I am going to die."
After five months of treatment in China and then in a clinic in Nevada, O'Kelley was able to return home to her seven children for continued treatment by a physician in Houston. Norris shared their tax return records documenting $2 million over three years in uninsured medical expenses to help O'Kelley return to health. Now Norris is suing 11 medical companies for the part they played in not warning the couple and others of the dangers of using a GBCA for MRI contrast.18
O'Kelley suffered confusion, muscle spasms, kidney damage and muscle wasting from a heavy metal contrast agent her doctors told her would be cleared from her body within hours after the MRI. The couple's attorney, Todd Walburg, told CBS News,19 "We have clients who have been misdiagnosed with Lyme disease, ALS, and then they've eventually ruled all those things out and the culprit remaining is the gadolinium."
Although the FDA has been aware of a strong association between gadolinium and kidney damage since 2006,20 and are aware GBCAs may deposit in organ and brain tissue,21 the agency insists the contrast dye is safe for use22 but states it will continue to assess safety. Norris states their intention is to draw attention to the problem and hopefully help others who suffer from the physical effects of the contrast agent.
Gadolinium Linked to Several Health Problems
In a comprehensive review of previous studies, researchers evaluated the effects of gadolinium and found that although the heavy metal was bound to chelating agents designed to help the chemical be eliminated from the body, gadolinium was found deposited in organs, bone and brain tissue.23 Their findings contradict statements from the FDA and pharmaceutical companies that only those with previous history of kidney damage may experience tissue deposits. Lead author and toxicologist Dr. Stacy Branch said:24
"Given the ever-growing toxicological and gadolinium tissue retention data, it is vital that the FDA promptly leads efforts, including retrospective and prospective clinical studies, to better define the connection between GBCA-exposure and adverse health events. This is needed to guide the choice of preventive methods, achieve accurate diagnoses, implement effective treatment approaches, and spark research for the design of safer contrast agents and imaging protocols."
These results were supported in a recent study from Case Western University, where researchers found gadolinium deposits in brain tissue of people who underwent more than one MRI with GBCA.25 The challenge faced by patients is that the symptoms they experience from the GBCA often result in the recommendation for another MRI with contrast to diagnose the problem. Repeated doses increase the risk for further health damage.
It seems ludicrous to imagine that deposits of heavy metal in your bone, brain and organs would not cause cognitive problems or other health damage, but that is exactly what the FDA is saying, as they acknowledge gadolinium is deposited but state they have no evidence these deposits are dangerous to your health.26
Recent research has also shown GBCA more easily passes the blood brain barrier in individuals who have neurological disease such as MS, stroke or a brain tumor.27 However, these neurological conditions are often a reason an MRI with GBCA is ordered, increasing the risk for further neurological damage to these individuals.
Support Your Body's Efforts to Eliminate Heavy Metal
It is vital you carefully review the recommendation to undergo an enhanced MRI with your physician and get a second opinion if you aren't completely satisfied. It is important to remember that you are the one who will experience the consequences of GBCA use and not your physician, radiologist or the FDA. If an MRI with contrast is absolutely necessary, there are several steps you can take that may help reduce gadolinium deposits and help your body detoxify.
Many of these steps will also help detoxify your body of other heavy metals and improve your overall health. Remember, if you do experience symptoms, a second MRI with contrast will only intensify the problem and not diagnose the issue. Seek out the care of an integrative medicine physician who can help guide your efforts to detoxify and consider the following steps:28,29
Optimize your gut microbiome
When your gut is functioning optimally without inflammation or leaking, toxins may be eliminated through your liver and gastrointestinal (GI) tract, which reduces the toxic load on your kidneys. Eliminate foods that commonly cause inflammation in your gut, such as wheat, refined sugar and corn.
Reduce your carbohydrate intake to 50 grams or less per day and eat fermented foods to help colonize your gut with beneficial bacteria. Add bone broth to your nutritional plan to help heal a leaky gut.
Optimize your nutrition
Healthy omega-3 fats, zinc and selenium help support your body during detoxification. Drink enough fresh, pure water so your urine is a light straw color to support your kidneys. Eat foods high in fiber to feed your beneficial bacteria. Broccoli, kale, garlic and onions support your liver during detoxification and add fiber to your diet.
Eat more fermented foods
Traditionally fermented food usually contains at least one cruciferous vegetable. Studies have now shown these vegetables are high in sulforaphane that protects your cells from inflammation30 and promotes healing and protection of the blood-brain barrier.31
Diatomaceous earth develops from fossilized shells of freshwater diatoms. It is found in large quantities across the earth. However, in order to use it on yourself or your pets it must be food grade. Nonfood grade is used in swimming pool filters and as an insecticide in around nonfood plants. Food grade diatomaceous earth binds heavy metals and other toxins in your GI tract, gently helping them pass out of your body.32
According to the Weston A. Price Foundation, you may dissolve less than 1 teaspoon for every 100 pounds of body weight in a glass of fresh, pure water and drink just before bed. Over a period of weeks or months, gradually increase this amount to nearly 1 tablespoon. This gradual increase will help reduce any GI discomforts.
Used this way, the metal binding capacity is unlikely to hinder the absorption of other needed minerals, such as calcium, magnesium or zinc that are consumed during the day.
Sweating in a sauna may help eliminate heavy metals and other toxins such as BPA.33 Anytime you use a sauna, carefully clean the area where you are seated as toxins that were eliminated from the previous use could be absorbed as your pores open and you begin to sweat.
Cilantro and chlorella have long been used to help detoxify from heavy metals.34 Cilantro mobilizes toxins from your tissues, often faster than your body can eliminate them. Chlorella helps to bind the metals and improve elimination, thus reducing the possibility of experiencing the effects of retoxification when heavy metals have been mobilized but not removed.
The Lighthouse Project Gadolinium Toxicity35 gathers information from individuals who have been poisoned by gadolinium and the treatments they have used. While chelation seems to be a logical choice, when used alone they have no record of individuals experiencing a complete remission of symptoms, so be sure to implement a more comprehensive, holistic plan.
Medical chelation is a process where a chemical agent is administered that binds to the metal, making it a stable compound that can be excreted. Chemical agents can be administered orally, through an IV or rectally.
Brain Regeneration: Can Infrared Light Reverse Parkinson’s and Alzheimer’s?
Posted on: Tuesday, November 21st 2017 at 12:30 pm
This article is copyrighted by GreenMedInfo LLC, 2017
Contrary to conventional wisdom, brain regeneration is possible. One promising therapy that promotes neurogenesis and is effective in pre-clinical studies of Alzheimer’s and Parkinson’s is near infrared light therapy, and it may improve other mental illnesses and neurodegenerative disorders including dementia, stroke, ALS, and traumatic brain injury as well.
Alzheimer’s disease and Parkinson’s disease are the most common neurodegenerative disorders. The former is a type of dementia that occurs secondary to the accumulation of abnormal protein deposits in the brain, including β-amyloid plaques and intraneuronal neurofibrillary tangles made of tau protein (1). Upon neuroimaging studies, gross cerebral cortical atrophy is found, meaning that the part of the brain responsible for executive functions such as learning, memory, language, decision-making, and problem-solving progressively degenerates (1). In addition, gliosis, or brain inflammation, is a hallmark characteristic of Alzheimer’s (1).
One hypothesis that is championed proposes that Alzheimer’s occurs due to self-propagating, prion-like protein assemblies, which interfere with the function of nerve cells (2). An alternate theory is that these so-called proteinopathies occur secondary to a microvascular hemorrhage or brain bleed (3). The brain bleed is believed to be the result of age-induced degradation of cerebral capillaries, which creates neuron-killing protein plaques and tangles (3).
Dysfunction of mitochondria, the energy-generating powerhouses of the cell, is also implicated in Alzheimer’s, as reduced efficacy of these organelles creates oxidative stress-inducing reactive oxygen species, or free radicals, which lead to neuronal cell death (4). Whatever the cause, extensive death of brain cells occurs, which explains the cognitive deficits that occur with Alzheimer’s disease, in addition to symptoms such as impaired judgment, confusion, agitation, linguistic abnormalities, social withdrawal, and even hallucinations (1).
Parkinson’s disease, on the other hand, is characterized by progressive death of dopamine-producing neurons in a region of the brainstem called the substantial nigra, but it can extend to other brain areas such as the locus coeruleus, olfactory bulb, dorsal motor nucleus of the vagal nerve, and even the cortex in late stages (5). As a result, the primary manifestation is that dopamine deficiency appears in the basal ganglia, a set of nuclei embedded deep in the brain hemispheres that is responsible for motor control (6). This leads to the cardinal manifestation of Parkinson’s, namely, a movement disorder that includes bradykinesia or slow movement, loss of voluntary movement, muscular rigidity, and resting tremor (7).
Not unlike what happens in Alzheimer’s, accumulation of abnormal intracellular protein aggregates known as Lewy bodies, composed of a protein called α-synuclein, is thought to be central to the pathogenesis of Parkinson’s disease (8). Like Alzheimer’s, mitochondrial dysfunction induced by genetic mutations, toxic agents, or damage to blood vessels is also considered to contribute to neuron cell death in Parkinson’s (9). Toxin exposure is especially implicated, as animal studies hint that development of Parkinson’s disease may occur as a byproduct of exposure to neurotoxins such as rotenone or paraquat (10). Impaired blood brain barrier function and damage to the endothelial cells of the vascular system, which line the interior surface of blood vessels, are also thought to play a role in Parkinson’s (10).
Overturning Old Notions of Neuroscience
The central dogma of neuroscience conceived of the central nervous system tissue as “perennial” after the doctrines of Giulio Bizzozero, the most prominent Italian histologist, who decreed that the lifelong cells of the nervous system were devoid of replicative potential (11). In other words, the perennial nature ascribed to the nerve cells of the brain and spinal cord meant that nerve cells were believed to be incapable of undergoing proliferation, or cell division, in the postnatal brain (11). While the early stage of in utero prenatal development known as embryogenesis permits massive neurogenesis, or the ability to create new nerve cells, the scientific consensus up until the end of the twentieth century held that neurogenesis was arrested after birth in mammals.
Santiago Ramon y Cajal, who led the charge in the neuroscience discipline in the later half of the nineteenth century onward and won a Nobel Prize for Medicine and Physiology, in fact stated that: “Once development was ended, the fonts of growth and regeneration of the axons and dendrites dried up irrevocably. In adult centers, the nerve paths are something fixed and immutable: everything may die, nothing may be regenerated” (11). Acknowledgment of the mere possibility of adult neurogenesis was hampered by the fact that scientists lacked the visualization techniques to detect neural stem cells, the precursors to new neurons and means by which neurogenesis occurs, and also did not have access to the molecular markers and microscopy required to observe cells in different cycle phases.
This view of nervous tissue as perennial was also reinforced by clinical observations that patients with chronic neurodegeneration, traumatic brain lesions, and cerebrovascular diseases do not experience functional recovery (11). Prevailing theories posited that adult neurogenesis was an evolutionary unlikelihood, since it would interfere with pre-existing neuronal connections and the fine-tuned electrochemical communication in the nervous system, as well as disrupt memory recall, which was believed to occur via stable neuronal circuits created and encoded during learning (11).
That brain cells are finite, and incapable of regeneration, painted a portrait of doom and gloom and inexorable debilitation for patients suffering from devastating neurodegenerative conditions. However, relatively recent discoveries have overturned these antiquated conceptions by revealing that the brain is plastic, or pliable, and that even neurons in adult higher vertebrates are capable of neurogenesis.
Scientists Discover Neural Regeneration is Possible
In the 1960s, these postulates of the old neurobiology were disproven when Joseph Altman and colleagues performed an experiment where radioactively labelled thymidine, one of the nucleotide base pairs that makes up DNA, was incorporated into a brain area called the dentate gyrus of the hippocampus and integrated into the genetic material of what was later confirmed via electron microscopy to be dividing neurons (12, 13). In essence, this illustrated that neurons were undergoing mitosis, a process of cell division where genetically identical daughter cells are created, and showed that adult neurogenesis is possible.
Another nail in the coffin of this antiquated perception of the nervous system was that neural stem cells, the multipotent, self-renewing progenitors from which new neurons arise, were found in the brains of adult mammals, and discovered to undergo expansion in their populations when prompted by signaling molecules called growth factors and morphogens (11). The multiplication and differentiation of neural stem cells, which are residents of the central nervous system, is essential for neurogenesis (14). Neural stem cells are capable of generating all of the cell types of the nervous system, including astrocytes, glial cells, and what are called oligodendrocytes in the central nervous system and Schwann cells in the peripheral nervous system (11). Researchers Colucci-D’Amato and Bonita in fact state that, “To date neural stem cells have been isolated from nearly all areas of the embryonic brain and in a growing list of adult mammalian brain areas, including cerebellum and cortex” (11, p. 268).
Other advances, such as confocal microscopy and the identification of cellular markers which allowed the phenotype of cells to be characterized all culminated in the realization that neurogenesis occurs continuously in some brain area, such as the hippocampus and subventricolar zone (SVZ), the former of which is responsible for the formation and consolidation of memories (11). To date, neurogenesis has been shown to be influenced by various chemical, pharmacological, and environmental stimuli. For instance, work by researcher Fernando Nottebohm demonstrated the spontaneous replacement of neurons in the adult avian brain (15). In song birds such as canaries, which experience seasonal modification in their songs, new neurons are recruited into their neuronal circuitry in a way that may be dependent upon social and reproductive interactions, territorial defense, migratory patterns and food caching (15).
This all should serve as a beacon of hope for patients experiencing the ravages of neurodegenerative disease, as it may mean that epigenetics, or the way gene expression changes based on lifestyle factors, may lend itself to neurogenesis and the reversal of these scourges of mankind. For example, researchers state that an enriched environment, learning, exercise, exposure to different odorant molecules, and drugs such as antidepressants, steroids, and alcohol can all favorably or unfavorably impact neurogenesis (11). These newfound revelations are being used in fact as an impetus to find cures for a laundry list of neurodegenerative diseases (11).
Novel Therapy Shown to Grow New Nerve Cells
Despite this research, the prevailing view of neurodegenerative diseases such as Alzheimer’s and Parkinson’s is that their underlying pathophysiology, a relentless progression of neuronal death, remains irreversible (10). Thus far, then, approaches have aimed to slow or stop neuronal cell death or to develop disease-modifying treatments that could stabilize the rate of neurodegeneration (10). One non-pharmacological therapy that may be able to actually regenerate brain cells, however, is light in the near infrared range, also known as low-level laser or light emitting diode (LED) therapy that utilizes wavelengths in the red to infrared spectrum.
Near infrared light therapy has the potential to “mitigate ubiquitous processes relating to cell damage and death,” and may have applications in conditions that “converge on common pathways of inflammation and oxidative stress” (10). This is demonstrated by the widespread efficacy of near infrared light therapy in improving conditions including traumatic brain injury, ischemic stroke, major depression, and age-related macular degeneration (10). In traumatic brain injury, for example, treatment with near infrared light improves social, interpersonal, and occupational functions, reduces symptoms of post-traumatic stress disorder (PTSD), and is helpful for sleep (16).
Because near infrared light treatment improves cognitive and emotional dimensions (17) and enhances short-term memory and measures of sustained attention (18), researchers have long suspected its potential for neuropsychological disorders. In a revolutionary publication, scientists propose that infrared light is superior to pharmacological standard of care for these debilitating conditions given its neuron-saving abilities (10).
For instance, in mouse models of traumatic brain injury, near infrared light increases levels of brain-derived neurotrophic factor (BDNF), a protein which helps dying nerve cells survive (19). In addition, infrared light both improves neurological performance and increases the numbers of neuroprogenitor cells, the precursors to new neurons, in areas of the brain such as the dentate gyrus of the hippocampus and the sub ventricular zone (20).
Near Infrared Light Therapy in Alzheimer’s and Parkinson’s
Although human trials have not been yet conducted in Alzheimer’s disease, mouse studies show that near infrared treatment reduces its characteristic proteinopathies, decreasing brain levels of β-amyloid plaques and neurofibrillary tangles of tau proteins, while also ameliorating cognitive deficits (10). Cellular energy production, as indicated by levels of ATP, were increased in these studies alongside bolstered mitochondrial function and (10). In transgenic mouse models of Alzheimer’s, application of non-thermal near infrared light reversed significant deficits in working memory and significantly improved cognitive performance (21).
In animal models of Parkinson’s, near infrared treatment has been shown to rescue dopaminergic neurons, the subset that degenerate in this condition, from death (10). In addition, near infrared light treatment corrects the abnormal firing activity of neurons in deep subthalamic brain regions that occurs in parkinsonian conditions (22). Various animal models of Parkinson’s disease shown improved motor control and locomotor activity, as measured by both mobility and velocity, after near infrared is applied (10).
In a macaque monkey model of Parkinson’s, an optical fiber device that administered near infrared to the midbrain largely prevented the development of clinical signs of Parkinson’s when the animals were injected with a chemical known to induce this disorder (23). It also preserved a greater number of dopaminergic nigral cells compared to the monkeys that had not received infrared treatment (23). Limited case reports in humans have shown that near infrared administered through an intranasal apparatus improves symptoms in the majority of Parkinson’s patients, and that its application to the back of the head and upper neck reduced signs of Parkinson’s in one patient (10). Other reports indicate that gait, speech, cognitive function, and freezing episodes were improved in late-stage Parkinson’s patients who undertook this therapy (24), but the study was low-quality (10).
Mechanism of Action: How Near Infrared Promotes Neurogenesis
The ways in which near infrared promotes neurogenesis are multi-fold. There is evidence that near infrared light exerts a hormetic effect, acting as an adaptive or positive stressor. Another example of a hormetic effect is that exhibited by phytonutrients in fruits and vegetables, which act as antioxidants by paradoxically stimulating oxidative damage via a pro-oxidant mechanism. This in turn up-regulates our endogenous antioxidant defense system. Similarly, near infrared light activates cellular stress response systems by targeting a key enzyme in the electron transport chain which is responsible for mitochondrial-based energy production called cytochrome c oxidase, an enzyme that is fundamental to the cellular bioenergetics of nerve cells (25).
By accepting light in the near infrared range of the electromagnetic spectrum, this enzyme induces a change in the electrochemical potential of the mitochondrial membrane, jump-starting production of the cellular energy currency called adenosine triphosphate (ATP) and causing a mild burst in the synthesis of reactive oxygen species (ROS) (10). As a result, downstream signaling pathways are triggered which induce reparative and neuroprotective mechanisms, including neurogenesis, the creation of new synapses, and brain-based antioxidant and metabolic effects (25).
Restoration of mitochondrial function in the endothelial cells lining cerebral blood vessels may also help neurons survive by repairing the blood-brain barrier and vascular network which is compromised in neurogenerative conditions (10). Impressively, “This modulation of multiple molecular systems appears capable of both conditioning neurons to resist future damage and accelerating repair of neurons damaged by a previous or continuing insult” (10).
On the other hand, the application of near infrared light has been shown to elicit systemic effects, possibly via circulating molecular factors (10). In other words, light in the near infrared spectrum applied to a local area elicits benefits in distal tissues remote from the initial site, perhaps by stimulating immune cells that have a neuroprotective role (10). Another way in which near infrared light activates global effects in the body is by up-regulating the production of signaling molecules known as anti-inflammatory cytokines, while down-regulating pro-inflammatory cytokines (26).
Near infrared also mobilizes tissue repair processes by improving the migration of white blood cells to wounds, increasing neovascularization, or the formation of new blood vessels, and facilitating formation of collagen (27). There is also evidence that near-infrared light exposure causes stem cells from the bone marrow to navigate to the site of damage and to release so-called trophic factors such as BDNF, which enhances nerve cell function and survival (28). Lastly, a system of communication between the mitochondria in the brain and the mitochondria in the tissues may be at play, so that application of near infrared light at a point in the body far from the brain can lead to neural regeneration (10).
Practical Application of Near Infrared Light Therapy
The key to mitigating the burden of chronic illness lies in physiological regeneration, which is emerging as a physiological inevitability, even in regions of the body where it was previously not thought possible. The ability to regenerate, secondary to normal biological processes of cellular erosion and decay, is programmed into our body in order for us to regain homeostasis.
So-called “photobiomodulation,” which includes near infrared light therapy, has limitless possible applications, and has even been shown to improve animal models of wound healing, heart attack, spinal cord injury, stroke, arthritis, familial amylotropic lateral sclerosis (FALS), diabetic ulcers, carpal tunnel syndrome, major depression, generalized anxiety disorder, frontotemporal dementia (29) and traumatic brain injury (27).
The biggest obstacle with infrared light therapy in neurodegenerative disease is targeting the zone of pathology, “when there are many intervening body tissues, namely skin, thick cranium, and meninges, and brain parenchyma,” since there is considerable dissipation of the signal across each millimeter of brain tissue (10). This is less problematic in Alzheimer’s, where the target regions are more superficial structures, but less easily rectified in the case of Parkinson’s, where there is significant distance from cranium to the brainstem where neurodegeneration takes place (10).
With Alzheimer’s, optimal delivery would be a near infrared light-emitting helmet worn over the entire cranium (10). Parkinson’s patients can achieve symptomatic relief when near infrared is applied in this fashion, as this would influence the abnormal neural circuitry in the cortex. However, to circumvent the problem of the sheer distance to the region of pathology in the brainstem, researchers propose that the minimally invasive surgical implantation of an optical fiber device near the brain parenchyma would be ideal, which would deliver therapeutic levels of near infrared (10). Until these options are commercially available, photobiomodulation devices or near infrared saunas may be a viable option, although human studies have not proved their efficacy.
Given its large margin of safety and lack of adverse effects, near infrared light therapy should be offered as an option for patients suffering from a myriad of chronic conditions, but is especially promising for neurodegenerative diseases including Alzheimer’s and Parkinson’s and may even have future use in multiple sclerosis. Near infrared therapy is superior to the mainstay drug treatments for these diseases since pre-clinical studies have demonstrated proof-of-concept that near infrared either arrests or slows the underlying pathology of these disease processes, and leads to the birth of new neurons, rather than merely mitigating symptoms (10).
1. Bird, T.D. (1998). Alzheimer disease overview. GeneReviews® [Internet]. Retrieved from https://www.ncbi.nlm.nih.gov/books/NBK1161/
2. Goedert, M. (2015). Alzheimer's and Parkinson's diseases: the prion concept in relation to assembled Aβ, tau, and α-synuclein. Science, 349, 1255555.
3. Stone, J. (2008). What initiates the formation of senile plaques? The origin of Alzheimer-like dementias in capillary haemorrhages. Medical Hypotheses, 71, 347–359.
4. Gonzalez-Lima, F., Barksdale B.R., & Rojas J.C. (2014). Mitochondrial respiration as a target for neuroprotection and cognitive enhancement. Biochemical Pharmacology, 88, 584–593. 10.1016/j.bcp.2013.11.010
5. Bergman, H., & Deuschl, G. (2002). Pathophysiology of Parkinson's disease: from clinical neurology to basic neuroscience and back. Movement Disorders, 7(Suppl. 3), S28–S40.
6. Lanciego, J.L., Luquin, N., & Obeso, J.A. (2012). Functional Neuroanatomy of the Basal Ganglia. Cold Springs Harbor Perspectives in Medicine, 2(12), a009621.
7. De Virgilio, A. et al. (2016). Parkinson's disease: Autoimmunity and neuroinflammation. Autoimmunity Reviews, 15(10), 1005-1011. doi: 10.1016/j.autrev.2016.07.022.
8. Gitler A.D. et al. (2009). Alpha-synuclein is part of a diverse and highly conserved interaction network that includes PARK9 and manganese toxicity. Natural Genetics, 41, 308–315.
9. Exner, N. et al. (2012). Mitochondrial dysfunction in Parkinson's disease: molecular mechanisms and pathophysiological consequences. EMBO Journal, 31, 3038–3062. 10.1038/emboj.2012.170
10. Johnstone, D.M. et al. (2015). Turning On Lights to Stop Neurodegeneration: The Potential of Near Infrared Light Therapy in Alzheimer's and Parkinson's Disease. Frontiers in Neuroscience, 9, 500. doi: 10.3389/fnins.2015.00500
11. Colucci-D’Amato, L., & Bonavita, V. (2006). The end of the central dogma of neurobiology: stem cells and neurogenesis in adult CNS. Neurological Science, 27(4), 266-270.
12. Altman, J. (1962). Are new neurons formed in the brains of adult mammals? Science, 135, 1127-1128.
13. Kaplan, M.S., & Hinds, J.W. (1977). Neurogenesis in the adult rat: electron microscopic analysis of light radioautographs. Science, 197, 1092-1094.
14. Martino, G. et al. (2011). Brain regeneration in physiology and pathology: the immune signature driving therapeutic plasticity of neural stem cells. Physiological Reviews, 91(4), 1281-1304.
15. Nottebohm, F. (2002). Why are some neurons replaced in adult brain? Journal of Neuroscience, 22(3), 624-628.
16. Naeser, M.A. et al. (2014). Significant improvements in cognitive performance post-transcranial, red/near-infrared light-emitting diode treatments in chronic, mild traumatic brain injury: open-protocol study. Journal of Neurotrauma, 31,(11), 1008-1017. doi: 10.1089/neu.2013.3244.
17. Barrett, D.W., & Gonzalez-Lima, F. (2013). Transcranial infrared laser stimulation produces beneficial cognitive and emotional effects in humans. Neuroscience, 230, 13-23. doi: 10.1016/j.neuroscience.2012.11.016.
18. Blanco, N.J., Maddox, W.T., & Gonzalez-Lima, F. (2015). Journal of Neuropsychology, 11(1),14-25. doi: 10.1111/jnp.12074.
19. Xuan, W. et al. (2013). Transcranial low-level laser therapy improves neurological performance in traumatic brain injury in mice: effect of treatment repetition regimen. PLoS ONE, 8, e53454.
20. Xuan, W. et al. (2014). Transcranial low-level laser therapy enhances learning, memory, and neuroprogenitor cells after traumatic brain injury in mice. Journal of Biomedical Optics, 191(10), 108003.
21. Michalikova, S. et al. (2008). Emotional responses and memory performance of middle-aged CD1 mice in a 3D maze: effects of low infrared light. Neurobiology of Learning and Memory, 89(4), 480-488.
22. Shaw, V.E. et al. (2012). Patterns of Cell Activity in the Subthalamic Region Associated with the Neuroprotective Action of Near-Infrared Light Treatment in MPTP-Treated Mice. Parkinsonian Disease, 2012, 29875. doi: 10.1155/2012/296875.
23. Darlot, F. et al. (2016). Near-infrared light is neuroprotective in a monkey model of Parkinson disease. Annals of Neurology, 79(1), 59-65. doi: 10.1002/ana.24542.
24. Maloney, R., Shanks, S., & Maloney J. (2010). The application of low-level laser therapy for the symptomatic care of late stage Parkinson's disease: a non-controlled, non-randomized study. American Society of Laser Medicine and Surgery, 185.
25. Rojas, J.C., & Gonzalez-Lima, F. (2011). Low-level light therapy of the eye and brain. Eye and Brain, 3, 49–67.
26. Muili, K.A. et al. (2012). Amelioration of experimental autoimmune encephalomyelitis in C57BL/6 mice by photobiomodulation induced by 670 nm light. PLoS ONE, 7, e30655.
27. Chung, H. et al. (2012). The Nuts and Bolts of Low-level Laser (Light) Therapy. Annals of Biomedical Engineering, 40(2), 516-533.gma
28. Hou, S.T. et al. (2008). Permissive and Repulsive Cues and Signalling Pathways of Axonal Outgrowth and Regeneration. International Review of Cell and Molecular Biology, 267, 121-181.
29. Purushothuman, S. et al. (2013). The impact of near-infrared light on dopaminergic cell survival in a transgenic mouse model of parkinsonism. Brain Research, 1535, 61–70.
Ali Le Vere holds dual Bachelor of Science degrees in Human Biology and Psychology, minors in Health Promotion and in Bioethics, Humanities, and Society, and is a Master of Science in Human Nutrition and Functional Medicine candidate. Having contended with chronic illness, her mission is to educate the public about the transformative potential of therapeutic nutrition and to disseminate information on evidence-based, empirically rooted holistic healing modalities. Read more at @empoweredautoimmune on Instagram and at www.EmpoweredAutoimmune.com: Science-based natural remedies for autoimmune disease, dysautonomia, Lyme disease, and other chronic, inflammatory illnesses.
Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of GreenMedInfo or its staff.
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Tuesday, November 21, 2017
Sunspots and Cell Towers Fueled the Northern California Firestorm
Forest fire scarred mountain slopes in the Colorado Rockies, where pine trees were stripped bare by the fire. [Credit: James Grundvig, August 11, 2017.]
Two seismic, overlapping events — one celestial, the other manmade — created the unique condition igniting the Northern California fires that swept through wine country on October 8 and 9. The chain reaction setoff more than a dozen clusters at the same time scattered across multiple counties on one hellish night.
At first, authorities suspected arson. Journalists absurdly blamed ISIS, then Mexican drug cartels for coordinating attacks. Napa and Sonoma Counties are not the regions to grow cannabis; pot farms are located far north along the coast.
The wildfires weren’t wild at all. The houses cooked from the inside out. They often burned from the top of the roofs, incinerating the walls, floors, appliances, and cladding down to the foundation. Yet, 90 percent of the trees on the properties remained standing with leaves on the branches — not burnt, merely dried out. It was as if a neutron bomb had detonated burning everything manmade, but leaving the greenery alone.
Drone footage, photos, and eyewitness accounts confirmed that was the case. Pine trees trapped by the fires stood unscathed, while the houses burned. Scarred shells of cars seen in photo after photos had their tires vaporized to the rims, the windshields melted away, and the metal bodies warped by the intense heat. None of it was normal or had been seen before.
What caused the inferno? How did it escalate so fast? Why did the fires burn so hot?
Wrong Theories Die Fast
With the houses bursting in flames on their own, the claim that the ‘Diablo’ wind caused the raging fires is patently false. The winds, on the still, cloudless night, came after the fires erupted. While “dry lightning,” an event that needs a cloud-to-ground funnel didn’t happen that evening.
Others have theorized that a military direct energy weapon (DEW) — aka laser — was used to ignite the structures on the ground in a microwave effect. Contrary to the fantasy of James Bond’s Goldeneye, however, the limitation of laser-mounted weapons has a maximum range of 60 miles, but mostly far less than five miles. They won’t be combat operational until the next couple of years.
One long time veteran Fire Captain John Lord, in the Lake County Region north of Napa, went on record in a video interview that a DEWs weapon started the fires and that “plastics” in modern home construction materials were the cause. Fire Captain Lord is close on both the ignition and the flammable material, but not quite there.
Solar Flares and Hurricanes
The 1859 coronal mass ejection (CME), the largest in history, lasted five days. The direct hit, known as the Carrington Flare, penetrated the earth’s magnetosphere sending “electric shocks through telegraphs lines” and “threw sparks” at the poles.
This century, scientists have worried that a CME event would wipe out communication satellites. But like the Carrington Flare, they failed to look closer to earth. What would the damage be if sunspots breached the magnetosphere? In 2017, after an active summer of solar flares, governments need to focus on terrestrial hot spots and exposure, and worry less about GPS and satellite relays.
This past March, a pair of Russian scientists published a whitepaper, Evolution of Extra-tropical Cyclones During Disturbed Geomagnetic Conditions.
It concluded: “During geomagnetic disturbances, favorable conditions for increasing intensity and cyclone lifetime are formed . . . The distributions of the temperature of air masses of extratropical cyclones were shown to change due to the changes in geomagnetic activity.”
In October, they posted a video on YouTube analyzing this year’s high number of powerful hurricanes, from Harvey and Jose, to Maria and Ophelia. The last storm turned back toward Ireland becoming the “strongest east hurricane ever recorded.”
Contrary to Climate Change fueling the ferocity of hurricanes this season or in years past, it appears solar flares are the mechanism for both steering the hurricanes and their robustness. In matching the formation and landing of Ophelia with a six-day geomagnetic storm of a “coronal hole high-speed solar wind streams,” the scientists had the smoking gun. With each one of the major hurricanes this season, they matched the storm dates with solar flare activity.
They back-tested their theory on hurricanes Sandy (2012) and Katrina (2005), and one in 1974, each lining up with solar disturbances. At the 13:30 mark of the video, they discuss sunspots melting power lines and causing an electrical explosion in Washington in 2015, while igniting a transformer fire in India in 2012.
The solar flares that penetrated earth’s magnetosphere were not wreaking havoc on the array of satellites orbiting earth, but were fueling hurricanes at sea and zapping the power grid on land.
Days Before the Firestorm
A professional, who lives and works in Sonoma, and who wants to remain anonymous, wrote in an email: “Two weeks before the fire, people were thirsty. They bought large amounts of water emptying the stores’ shelves.” Yet the air wasn’t hotter than historical averages for the region — but it did have one weird effect on people’s hair being “statically charged with electricity.”
When the firestorm burst that Sunday evening, witnesses claimed to have seen an “orange glow” behind and above the trees. They didn’t attribute the halos to the fires, but to an unknown anomaly in the air that felt charged.
The Santa Rosa Fire Department and other investigators have suspected power lines played a role in causing the fires, while others blamed smart meters. Yes to both, but they played a diminished one, acting more like a conduit to igniting some of the structures. The electrical grid, however, wasn’t the mechanism that lit the fires or charged the air with static electricity in the days before the catastrophe.
1) Trees remain standing; shell of a car microwaved; 2) Hilton in Santa Rosa burned from the top down and inside out, greenery left alone; 3) House fire burned from the roof down, trees untouched; 4) House incinerated to the foundation slab, trees in the background left unscathed.
Cell Towers Generate Microwaves
In crowdsolving the firestorm, a few journalists, scientists, and this author believe that the 4G-network of cell towers created an electromagnetic field that allowed the solar flares to cook the houses directly without running through the power lines or smart meters. In total, 77 cell towers burned up or were damaged; they were not the source that lit the fires, but the feeders.
Cell towers burned, the houses baked, the trees remained standing.
The current cell tower system in use is called WiMAX: Worldwide Interoperability for Microwave Access. The 4G-system sends out signals through panels or antennas in narrow beams from tower to tower, not aimed at the ground. But the network does emit non-ionizing radiation, which the industry qualifies as typically safe: “It causes some heating effect, but usually not enough to cause any type of long-term damage to tissue. Radio-frequency energy, visible light and microwave radiation are considered non-ionizing.”
All well and good. But tell that to the dozens of people who died and the more than 7,500 homes and businesses that were destroyed by the fires.
If you put tinfoil in a microwave oven it will spark with flames shooting about, giving off an orange halo glow. It’s not the metal that catches fire, but the aluminum oxide coating. When superheated by microwave energy the coating releases oxygen on the surface engorging the material to burn even hotter. This phenomenon is known as “field-activated combustion synthesis.” Like a Duraflame log, the self-sustaining oxygen fuels the flames as had been seen with the ceramics roof tiles and the metals used to build — materials that do not burn in a normal forest fire.
With the invisible microwave field formed by the many clusters of cell towers in and around Napa and Sonoma, and the sunspots lighting the charged air, it created the orange halos while igniting hundreds of homes at once like Roman candles.
On October 7, Spaceweather.com observed that a “minor stream of solar wind brushed Earth’s magnetic field” and that a geomagnetic storm would hit on October 11.
Other evidence arose when a “second source of light,” a band that stretched from Washington state down to Arizona, crossed the US in less than thirty minutes, captured by the GOES 16 satellite on October 9. The speaker notes the anomaly, while eliminating the sun and moon as suspects, since the August 21 solar eclipse took more than two hours to transit across country.
For the professional who described the strange days before the event, she returned on October 24 to hear an update in a Hyatt Hotel. She noticed her watch acting peculiar and captured the compass and second arm going haywire — aroused by electromagnetic disturbances still in the area.
An error occurred.
The new vulnerability of the Digital Age boils down to a mix of oxide-coated materials, clusters of cell towers, and the right climate and atmospheric condition for sunspots to reach earth to ignite microwave energized air. Meaning, the buildings and houses in Silicon Valley are just as exposed as any place in California, and any place in the world with similar developments.
Monday, November 20, 2017
Cell Phone-Induced Bodily Harm: How The Bees Can Help
2Posted on: Monday, November 20th 2017 at 10:15 am
Written By: Sayer Ji, Founder
This article is copyrighted by GreenMedInfo LLC, 2017
Did you know that your cell phone technically microwaves your brain? And did you know that natural substances have proven radioprotective properties that can reduce your risk of adverse exposures?
Cell phones and the communications infrastructure that makes them possible are ubiquitous today, making complete avoidance of their significant radiotoxicy next to impossible. Plenty of evidence already exists showing that cell phones emit a type of electromagnetic radiation -- in the microwave range -- capable of adversely affecting a wide range of organs, with the nervous system of those exposed perhaps most sensitive to its adverse effects. Below is a sampling of some of their adverse health effects as demonstrated in the biomedical literature:
- Liver Damage
- Interruption of Sleep
- REM Cycle Disruption
- Heart Damage
- Fetal Harm
- Head Tumors
- Kidney Damage
- Acoustic Neuroma
- Brain Wave Disruption
Provocative research indicates that the problems associated with cell phone radiation exposure are far more profound that previously believed. In fact, pregnant women may need to exercise additional caution in order to protect their unborn from adverse neurological effects associated with cell phone radiation exposure.
In a study entitled, "The influence of microwave radiation from cellular phone on fetal rat brain," and published in the journal Electromagnetic Biology and Medicine in 2012, researchers discovered that pregnant rats exposed to microwave radiation from cellular phones had fetuses whose brains showed signs of harm, as measured by enhanced oxidative stress and altered levels of neurotransmitters.
We also reported more recently on clinical research indicating that as little as 15 minutes of "talk time" can profoundly alter and disrupt brain wave activity intimately connectd to cognition, mood, and behavior: Brain Wave Warping Effect of Mobile Phones.
For a concise explanation of the mechanisms behind cell-phone induced damage, watch Dr. Chris Busby's video on the topic below:
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Natural, Evidence-Based Ways to Protect Against Cell Phone Radiation
Given the wide range of potential risks associated with cell phones, we have plumbed the depths of MEDLINE in search of research on natural substances capable of ameliorating cell-phone associated toxicities.
Surprisingly, the little known bee product known as propolis exhibits powerful protective action against cell-phone induced damage to a variety of organs, including the kidney, heart and brain.
Once believed to function merely as mortar for plugging up small holes in the bee hive, propolis is now understood to have powerfully protective properties, such as its antimicrobial activity. The bees even use it to mummify animals that make their way into the hive, e.g. lizards, that they can not physically remove before they undergo putrefaction. Propolis' infection-fighting properties, however, are only the tip of the iceberg when it comes to its potential beneficial effects.
While there are over 100 potential therapeutic applications of propolis documented in the biomedical literature, propolis' radioprotective properties are perhaps the most intensely investigated and well established. We have, in fact, indexed 15 such studies on its ability to reduce radiation-induced damage, including gamma radiation commonly associated with medical diagnostic and radiotherapy procedures which you can view here.
Other substances capable of protecting against the radiation specific to the mobile phone range include melatonin, EGCG (green tea polyphenol), ginkgo biloba and the glutathione precursor NAC. To view the studies click the image below.
For additional research on radioprotective substances, visit our page dedicated to the topic.
Sayer Ji is founder of Greenmedinfo.com, a reviewer at the International Journal of Human Nutrition and Functional Medicine, Co-founder and CEO of Systome Biomed, Vice Chairman of the Board of the National Health Federation, Steering Committee Member of the Global Non-GMO Foundation.
Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of GreenMedInfo or its staff.
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